QQ客服 官方微博 反馈留言

	Myricitrin Attenuates High Glucose-Induced Apoptosis through Activating Akt-Nrf2 Signaling in H9c2 Cardiomyocytes
	Zhang, Bin; Chen, Yaping; Shen, Qiang; Liu, Guiyan; Ye, Jingxue; Sun, Guibo; Sun, Xiaobo
	2016
卷号	21期号:7
关键词	hyperglycemia oxidative stress reactive oxygen species apoptosis H9c2 cell
ISSN号	1420-3049
DOI	10.3390/molecules21070880
URL标识	查看原文
收录类别	SCIE ; PUBMED
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/6383625
专题	中国医学科学院 北京协和医学院
推荐引用方式 GB/T 7714	Zhang, Bin,Chen, Yaping,Shen, Qiang,et al. Myricitrin Attenuates High Glucose-Induced Apoptosis through Activating Akt-Nrf2 Signaling in H9c2 Cardiomyocytes[J],2016,21(7).
APA	Zhang, Bin.,Chen, Yaping.,Shen, Qiang.,Liu, Guiyan.,Ye, Jingxue.,...&Sun, Xiaobo.(2016).Myricitrin Attenuates High Glucose-Induced Apoptosis through Activating Akt-Nrf2 Signaling in H9c2 Cardiomyocytes.,21(7).
MLA	Zhang, Bin,et al."Myricitrin Attenuates High Glucose-Induced Apoptosis through Activating Akt-Nrf2 Signaling in H9c2 Cardiomyocytes".21.7(2016).

个性服务

查看访问统计

相关权益政策

暂无数据

收藏/分享

除非特别说明，本系统中所有内容都受版权保护，并保留所有权利。