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	Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks.
	Ling, C; Huang, J; Yan, ZJ; Li, YJ; Ohzeki, M; Ishiai, M; Xu, DY; Takata, M; Seidman, M; Wang, WD
刊名	Cell Discov
	2016
卷号	Vol.2页码:16047
关键词	BLM Bloom syndrome FANCM Fanconi anemia RMI2 interstrand crosslinks replication
ISSN号	2056-5968
URL标识	查看原文
公开日期	[db:dc_date_available]
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/6071149
专题	湖南大学
作者单位	1.NIA, Genet Lab, NIH, Baltimore, MD 21224 USA 2.NIA, Lab Mol Gerontol, NIH, Baltimore, MD 21224 USA 3.Kyoto Univ, Dept Late Effects Studies, Radiat Biol Ctr, Lab DNA Damage Signaling, Kyoto, Japan 4.Hunan Univ, Inst Chem Biol & Nanomed, Coll Biol, Changsha 410082, Hunan, Peoples R China 5.Peking Univ, Sch Life Sci, State Key Lab Prot & Plant Gene Res, Beijing 100871, Peoples R China
推荐引用方式 GB/T 7714	Ling, C,Huang, J,Yan, ZJ,et al. Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks.[J]. Cell Discov,2016,Vol.2:16047.
APA	Ling, C.,Huang, J.,Yan, ZJ.,Li, YJ.,Ohzeki, M.,...&Wang, WD.(2016).Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks..Cell Discov,Vol.2,16047.
MLA	Ling, C,et al."Bloom syndrome complex promotes FANCM recruitment to stalled replication forks and facilitates both repair and traverse of DNA interstrand crosslinks.".Cell Discov Vol.2(2016):16047.

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