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	SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion
	Sansbury, F. H.; Flanagan, S. E.; Houghton, J. A. L.; Shen, F. L. Shuixian; Al-Senani, A. M. S.; Habeb, A. M.; Abdullah, M.; Kariminejad, A.; Ellard, S.; Hattersley, A. T.
刊名	DIABETOLOGIA
	2012
卷号	55期号:9
关键词	Fanconi-Bickel syndrome FBS GLUT2 Neonatal diabetes mellitus Permanent neonatal diabetes mellitus PNDM SLC2A2 TNDM Transient neonatal diabetes mellitus
ISSN号	0012-186X
URL标识	查看原文
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/4913982
专题	复旦大学上海医学院
推荐引用方式 GB/T 7714	Sansbury, F. H.,Flanagan, S. E.,Houghton, J. A. L.,et al. SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion[J]. DIABETOLOGIA,2012,55(9).
APA	Sansbury, F. H..,Flanagan, S. E..,Houghton, J. A. L..,Shen, F. L. Shuixian.,Al-Senani, A. M. S..,...&Hattersley, A. T..(2012).SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion.DIABETOLOGIA,55(9).
MLA	Sansbury, F. H.,et al."SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion".DIABETOLOGIA 55.9(2012).

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