SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion | |
Sansbury, F. H.; Flanagan, S. E.; Houghton, J. A. L.; Shen, F. L. Shuixian; Al-Senani, A. M. S.; Habeb, A. M.; Abdullah, M.; Kariminejad, A.; Ellard, S.; Hattersley, A. T. | |
刊名 | DIABETOLOGIA
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2012 | |
卷号 | 55期号:9 |
关键词 | Fanconi-Bickel syndrome FBS GLUT2 Neonatal diabetes mellitus Permanent neonatal diabetes mellitus PNDM SLC2A2 TNDM Transient neonatal diabetes mellitus |
ISSN号 | 0012-186X |
URL标识 | 查看原文 |
语种 | 英语 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/4913982 |
专题 | 复旦大学上海医学院 |
推荐引用方式 GB/T 7714 | Sansbury, F. H.,Flanagan, S. E.,Houghton, J. A. L.,et al. SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion[J]. DIABETOLOGIA,2012,55(9). |
APA | Sansbury, F. H..,Flanagan, S. E..,Houghton, J. A. L..,Shen, F. L. Shuixian.,Al-Senani, A. M. S..,...&Hattersley, A. T..(2012).SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion.DIABETOLOGIA,55(9). |
MLA | Sansbury, F. H.,et al."SLC2A2 mutations can cause neonatal diabetes, suggesting GLUT2 may have a role in human insulin secretion".DIABETOLOGIA 55.9(2012). |
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