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Folic acid supplementation repressed hypoxia-induced inflammatory response via ROS and JAK2/STAT3 pathway in human promyelomonocytic cells
Ma, Jing[1,2]; Zhen, Xiaozhou[2,3]; Huang, Xiaoyan[2]; Jiang, Xinwei[2,4]
2018
卷号53页码:40
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/4763957
专题暨南大学
作者单位1.[1]Guangdong Prov Key Lab Food Nutr & Hlth, Guangzhou, Guangdong, Peoples R China
2.[2]Sun Yat Sen Univ, Sch Publ Hlth, Dept Nutr, Guangzhou, Guangdong, Peoples R China
3.[3]Sun Yat Sen Univ, Affiliated Hosp 3, Guangzhou, Guangdong, Peoples R China
4.[4]Jinan Univ, Inst Sci & Technol, Dept Food Sci & Engn, 601 Huangpu Rd, Guangzhou 510632, Guangdong, Peoples R China
推荐引用方式
GB/T 7714
Ma, Jing[1,2],Zhen, Xiaozhou[2,3],Huang, Xiaoyan[2],et al. Folic acid supplementation repressed hypoxia-induced inflammatory response via ROS and JAK2/STAT3 pathway in human promyelomonocytic cells[J],2018,53:40.
APA Ma, Jing[1,2],Zhen, Xiaozhou[2,3],Huang, Xiaoyan[2],&Jiang, Xinwei[2,4].(2018).Folic acid supplementation repressed hypoxia-induced inflammatory response via ROS and JAK2/STAT3 pathway in human promyelomonocytic cells.,53,40.
MLA Ma, Jing[1,2],et al."Folic acid supplementation repressed hypoxia-induced inflammatory response via ROS and JAK2/STAT3 pathway in human promyelomonocytic cells".53(2018):40.
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