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Rosiglitazone elevates sensitization of drug-resistant oral epidermoid carcinoma cells to vincristine by G2/M-phase arrest, independent of PPAR-γ pathway
Wang H.-Y.; Zhang Y.; Zhou Y.; Lu Y.-Y.; Wang W.-F.; Xin M.; Guo X.-L.
刊名Biomedicine and Pharmacotherapy
2016
卷号83页码:349-361
关键词Cell cycle Cell proliferation and apoptosis Drug resistance reversal Oral epidermoid carcinoma PTEN/PI3K/AKT Rosiglitazone Vincristine
DOI10.1016/j.biopha.2016.06.047
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/4712978
专题山东大学
作者单位Department of Pharmacology, Key Laboratory of Chemical Biology , School of Pharmaceutical Sciences, Shandong Unive
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GB/T 7714
Wang H.-Y.,Zhang Y.,Zhou Y.,et al. Rosiglitazone elevates sensitization of drug-resistant oral epidermoid carcinoma cells to vincristine by G2/M-phase arrest, independent of PPAR-γ pathway[J]. Biomedicine and Pharmacotherapy,2016,83:349-361.
APA Wang H.-Y..,Zhang Y..,Zhou Y..,Lu Y.-Y..,Wang W.-F..,...&Guo X.-L..(2016).Rosiglitazone elevates sensitization of drug-resistant oral epidermoid carcinoma cells to vincristine by G2/M-phase arrest, independent of PPAR-γ pathway.Biomedicine and Pharmacotherapy,83,349-361.
MLA Wang H.-Y.,et al."Rosiglitazone elevates sensitization of drug-resistant oral epidermoid carcinoma cells to vincristine by G2/M-phase arrest, independent of PPAR-γ pathway".Biomedicine and Pharmacotherapy 83(2016):349-361.
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