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FAK contributes to proteinuria in hypercholesterolaemic rats and modulates podocyte F-actin re-organization via activating p38 in response to ox-LDL
Hu, Mengsi; Fan, Minghua; Zhen, Junhui; Lin, Jiangong; Wang, Qun; Lv, Zhimei; Wang, Rong
刊名JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
2017
卷号21期号:3页码:552-567
关键词podocyte hypercholesterolaemia ox-LDL FAK p38 cytoskeleton
DOI10.1111/jcmm.13001
URL标识查看原文
公开日期[db:dc_date_available]
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/4590312
专题山东大学
作者单位1.Shandong Univ, Shandong Prov Hosp, Dept Nephrol, Jinan, Shandong, Peoples R China.
2.[
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GB/T 7714
Hu, Mengsi,Fan, Minghua,Zhen, Junhui,et al. FAK contributes to proteinuria in hypercholesterolaemic rats and modulates podocyte F-actin re-organization via activating p38 in response to ox-LDL[J]. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE,2017,21(3):552-567.
APA Hu, Mengsi.,Fan, Minghua.,Zhen, Junhui.,Lin, Jiangong.,Wang, Qun.,...&Wang, Rong.(2017).FAK contributes to proteinuria in hypercholesterolaemic rats and modulates podocyte F-actin re-organization via activating p38 in response to ox-LDL.JOURNAL OF CELLULAR AND MOLECULAR MEDICINE,21(3),552-567.
MLA Hu, Mengsi,et al."FAK contributes to proteinuria in hypercholesterolaemic rats and modulates podocyte F-actin re-organization via activating p38 in response to ox-LDL".JOURNAL OF CELLULAR AND MOLECULAR MEDICINE 21.3(2017):552-567.
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