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	L-3-n-butylphthalide may attenuate A beta(1-42)-induced neuronal apoptosis and synaptotoxicity in primary cultured cortical neurons possibly via modulating both PI3K/AKT and ERK signaling pathways
	Zhao, Yunxia; Zhang, Yong; Chen, Xiuying; Dong, Wei; Du, Yifeng
刊名	INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE
	2018
卷号	11期号:9页码:9167-9177
关键词	Alzheimer's disease (AD) beta-amyloid (A beta) apoptosis PI3K/AKT pathway ERK pathway
URL标识	查看原文
公开日期	[db:dc_date_available]
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/4572565
专题	山东大学
作者单位	Shandong Univ, Shandong Prov Hosp, Dept Neurol, 324 Jingwu Rd, Jinan 250021, Shandong, Peoples
推荐引用方式 GB/T 7714	Zhao, Yunxia,Zhang, Yong,Chen, Xiuying,et al. L-3-n-butylphthalide may attenuate A beta(1-42)-induced neuronal apoptosis and synaptotoxicity in primary cultured cortical neurons possibly via modulating both PI3K/AKT and ERK signaling pathways[J]. INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE,2018,11(9):9167-9177.
APA	Zhao, Yunxia,Zhang, Yong,Chen, Xiuying,Dong, Wei,&Du, Yifeng.(2018).L-3-n-butylphthalide may attenuate A beta(1-42)-induced neuronal apoptosis and synaptotoxicity in primary cultured cortical neurons possibly via modulating both PI3K/AKT and ERK signaling pathways.INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE,11(9),9167-9177.
MLA	Zhao, Yunxia,et al."L-3-n-butylphthalide may attenuate A beta(1-42)-induced neuronal apoptosis and synaptotoxicity in primary cultured cortical neurons possibly via modulating both PI3K/AKT and ERK signaling pathways".INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE 11.9(2018):9167-9177.

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