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	ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington's disease models
	Wold, Mitchell S.; Lim, Junghyun; Lachance, Veronik; Deng, Zhiqiang; Yue, Zhenyu
刊名	MOLECULAR NEURODEGENERATION
	2016
卷号	11
关键词	ATG14 Vps34 ULK1 Autophagy Huntington's
ISSN号	1750-1326
DOI	10.1186/s13024-016-0141-0
URL标识	查看原文
收录类别	SCIE
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/4089150
专题	武汉大学
推荐引用方式 GB/T 7714	Wold, Mitchell S.,Lim, Junghyun,Lachance, Veronik,et al. ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington's disease models[J]. MOLECULAR NEURODEGENERATION,2016,11.
APA	Wold, Mitchell S.,Lim, Junghyun,Lachance, Veronik,Deng, Zhiqiang,&Yue, Zhenyu.(2016).ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington's disease models.MOLECULAR NEURODEGENERATION,11.
MLA	Wold, Mitchell S.,et al."ULK1-mediated phosphorylation of ATG14 promotes autophagy and is impaired in Huntington's disease models".MOLECULAR NEURODEGENERATION 11(2016).

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