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Sevoflurane-Induced Endoplasmic Reticulum Stress Contributes to Neuroapoptosis and BACE-1 Expression in the Developing Brain: The Role of eIF2 alpha
Liu, Bin; Xia, Junming; Chen, Yali; Zhang, Jun
刊名NEUROTOXICITY RESEARCH
2017
卷号31期号:2
关键词Sevoflurane ER stress eIF2 alpha Neuroapoptosis BACE-1 Developing brain
ISSN号1029-8428
URL标识查看原文
语种英语
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3620559
专题复旦大学上海医学院
推荐引用方式
GB/T 7714
Liu, Bin,Xia, Junming,Chen, Yali,et al. Sevoflurane-Induced Endoplasmic Reticulum Stress Contributes to Neuroapoptosis and BACE-1 Expression in the Developing Brain: The Role of eIF2 alpha[J]. NEUROTOXICITY RESEARCH,2017,31(2).
APA Liu, Bin,Xia, Junming,Chen, Yali,&Zhang, Jun.(2017).Sevoflurane-Induced Endoplasmic Reticulum Stress Contributes to Neuroapoptosis and BACE-1 Expression in the Developing Brain: The Role of eIF2 alpha.NEUROTOXICITY RESEARCH,31(2).
MLA Liu, Bin,et al."Sevoflurane-Induced Endoplasmic Reticulum Stress Contributes to Neuroapoptosis and BACE-1 Expression in the Developing Brain: The Role of eIF2 alpha".NEUROTOXICITY RESEARCH 31.2(2017).
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