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	Reduced nitric oxide bioavailability mediates cerebroarterial dysfunction independent of cerebral amyloid angiopathy in a mouse model of Alzheimer's disease
	Merlini, Mario; Shi, Yi; Keller, Stephan; Savarese, Gianluigi; Akhmedov, Alexander; Derungs, Rebecca; Spescha, Remo D.; Kulic, Luka; Nitsch, Roger M.; Luscher, Thomas F.
刊名	AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
	2017
卷号	312期号:2
关键词	Alzheimer's disease cerebral amyloid angiopathy cerebrovascular pathology endothelial dysfunction nitric oxide cyclic GMP
ISSN号	0363-6135
URL标识	查看原文
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3617336
专题	复旦大学上海医学院
推荐引用方式 GB/T 7714	Merlini, Mario,Shi, Yi,Keller, Stephan,et al. Reduced nitric oxide bioavailability mediates cerebroarterial dysfunction independent of cerebral amyloid angiopathy in a mouse model of Alzheimer's disease[J]. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY,2017,312(2).
APA	Merlini, Mario.,Shi, Yi.,Keller, Stephan.,Savarese, Gianluigi.,Akhmedov, Alexander.,...&Camici, Giovanni G..(2017).Reduced nitric oxide bioavailability mediates cerebroarterial dysfunction independent of cerebral amyloid angiopathy in a mouse model of Alzheimer's disease.AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY,312(2).
MLA	Merlini, Mario,et al."Reduced nitric oxide bioavailability mediates cerebroarterial dysfunction independent of cerebral amyloid angiopathy in a mouse model of Alzheimer's disease".AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY 312.2(2017).

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