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	Gap junctional intercellular communication and endoplasmic reticulum stress regulate chronic cadmium exposure induced apoptosis in HK-2 cells
	Ge, Zehe; Diao, Haipeng; Ji, Xiaoli; Liu, Qingping; Zhang, Xiaoyan; Wu, Qing
刊名	TOXICOLOGY LETTERS
	2018
卷号	288
关键词	Cadmium HK-2 cells GJIC ER stress AKT signaling
ISSN号	0378-4274
URL标识	查看原文
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3607930
专题	复旦大学上海医学院
推荐引用方式 GB/T 7714	Ge, Zehe,Diao, Haipeng,Ji, Xiaoli,et al. Gap junctional intercellular communication and endoplasmic reticulum stress regulate chronic cadmium exposure induced apoptosis in HK-2 cells[J]. TOXICOLOGY LETTERS,2018,288.
APA	Ge, Zehe,Diao, Haipeng,Ji, Xiaoli,Liu, Qingping,Zhang, Xiaoyan,&Wu, Qing.(2018).Gap junctional intercellular communication and endoplasmic reticulum stress regulate chronic cadmium exposure induced apoptosis in HK-2 cells.TOXICOLOGY LETTERS,288.
MLA	Ge, Zehe,et al."Gap junctional intercellular communication and endoplasmic reticulum stress regulate chronic cadmium exposure induced apoptosis in HK-2 cells".TOXICOLOGY LETTERS 288(2018).

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