Respiratory Syncytial virus infection compromises asthma tolerance by recruiting interleukin-17A-producing cells via CCR6-CCL20 signaling (vol 88, pg 45, 2017) | |
He, Yanchao; Sun, Wei; Wu, Yi; Li, Ling; Jie, Zhijun; Su, Xiao; Shi, Tianyun | |
刊名 | MOLECULAR IMMUNOLOGY
![]() |
2018 | |
卷号 | 93 |
ISSN号 | 0161-5890 |
URL标识 | 查看原文 |
语种 | 英语 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/3605863 |
专题 | 复旦大学上海医学院 |
推荐引用方式 GB/T 7714 | He, Yanchao,Sun, Wei,Wu, Yi,et al. Respiratory Syncytial virus infection compromises asthma tolerance by recruiting interleukin-17A-producing cells via CCR6-CCL20 signaling (vol 88, pg 45, 2017)[J]. MOLECULAR IMMUNOLOGY,2018,93. |
APA | He, Yanchao.,Sun, Wei.,Wu, Yi.,Li, Ling.,Jie, Zhijun.,...&Shi, Tianyun.(2018).Respiratory Syncytial virus infection compromises asthma tolerance by recruiting interleukin-17A-producing cells via CCR6-CCL20 signaling (vol 88, pg 45, 2017).MOLECULAR IMMUNOLOGY,93. |
MLA | He, Yanchao,et al."Respiratory Syncytial virus infection compromises asthma tolerance by recruiting interleukin-17A-producing cells via CCR6-CCL20 signaling (vol 88, pg 45, 2017)".MOLECULAR IMMUNOLOGY 93(2018). |
个性服务 |
查看访问统计 |
相关权益政策 |
暂无数据 |
收藏/分享 |
除非特别说明,本系统中所有内容都受版权保护,并保留所有权利。
修改评论