CORC  > 吉林大学白求恩第一医院
Compound K, a final intestinal metabolite of ginsenosides, enhances insulin secretion in MIN6 pancreatic beta-cells by upregulation of GLUT2
Gu, Jian; Li, Wei; Xiao, Dong; Wei, ShengNan; Cui, WanLi; Chen, WeiJia; Hu, YaLi; Bi, XiaoJia; Kim, YongChol; Li, Jing
刊名FITOTERAPIA
2013
卷号87
关键词Compound K Insulin secretion MIN6 cells GLUT2
ISSN号0367-326X
URL标识查看原文
语种英语
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3549090
专题吉林大学白求恩第一医院
推荐引用方式
GB/T 7714
Gu, Jian,Li, Wei,Xiao, Dong,et al. Compound K, a final intestinal metabolite of ginsenosides, enhances insulin secretion in MIN6 pancreatic beta-cells by upregulation of GLUT2[J]. FITOTERAPIA,2013,87.
APA Gu, Jian.,Li, Wei.,Xiao, Dong.,Wei, ShengNan.,Cui, WanLi.,...&Chen, Li.(2013).Compound K, a final intestinal metabolite of ginsenosides, enhances insulin secretion in MIN6 pancreatic beta-cells by upregulation of GLUT2.FITOTERAPIA,87.
MLA Gu, Jian,et al."Compound K, a final intestinal metabolite of ginsenosides, enhances insulin secretion in MIN6 pancreatic beta-cells by upregulation of GLUT2".FITOTERAPIA 87(2013).
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