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	(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.
	Sufang Liu; Hongde Li; Lin Chen; Lifang Yang; Lili Li; Yongguan Tao; Wei Li; Zijian Li; Haidan Liu; Min Tang
刊名	Carcinogenesis
	2013
卷号	34期号:3页码:627-637
ISSN号	0143-3334
DOI	10.1093/carcin/bgs364
URL标识	查看原文
WOS记录号	WOS:000315629000016
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3317929
专题	中南大学
作者单位	1.[Lifang Yang 2.Lin Chen 3.Haidan Liu 4.Lili Li 5.Wei Li 6.Min Tang 7.Sufang Liu 8.Zijian Li 9.Yongguan Tao 10.Hongde Li
推荐引用方式 GB/T 7714	Sufang Liu,Hongde Li,Lin Chen,et al. (-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.[J]. Carcinogenesis,2013,34(3):627-637.
APA	Sufang Liu.,Hongde Li.,Lin Chen.,Lifang Yang.,Lili Li.,...&Ya Cao.(2013).(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells..Carcinogenesis,34(3),627-637.
MLA	Sufang Liu,et al."(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.".Carcinogenesis 34.3(2013):627-637.

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