QQ客服 官方微博 反馈留言

	Non-canonical GLI1/2 activation by PI3K/AKT signaling in renal cell carcinoma: A novel potential therapeutic target
	Zhou, Jiancheng; Zhu, Guodong; Huang, Jun; Li, Lei; Du, Yuefeng; Gao, Yang; Wu, Dapeng; Wang, Xinyang; Hsieh, Jer-Tsong; He, Dalin
刊名	CANCER LETTERS
	2016
卷号	370期号:[db:dc_citation_issue]页码:313-323
关键词	Therapeutic target VHL PI3K/AKT Renal cell carcinoma Hedgehog pathway
ISSN号	0304-3835
DOI	[db:dc_identifier_doi]
URL标识	查看原文
WOS记录号	[DB:DC_IDENTIFIER_WOSID]
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3220998
专题	西安交通大学
推荐引用方式 GB/T 7714	Zhou, Jiancheng,Zhu, Guodong,Huang, Jun,et al. Non-canonical GLI1/2 activation by PI3K/AKT signaling in renal cell carcinoma: A novel potential therapeutic target[J]. CANCER LETTERS,2016,370([db:dc_citation_issue]):313-323.
APA	Zhou, Jiancheng.,Zhu, Guodong.,Huang, Jun.,Li, Lei.,Du, Yuefeng.,...&Wu, Kaijie.(2016).Non-canonical GLI1/2 activation by PI3K/AKT signaling in renal cell carcinoma: A novel potential therapeutic target.CANCER LETTERS,370([db:dc_citation_issue]),313-323.
MLA	Zhou, Jiancheng,et al."Non-canonical GLI1/2 activation by PI3K/AKT signaling in renal cell carcinoma: A novel potential therapeutic target".CANCER LETTERS 370.[db:dc_citation_issue](2016):313-323.

个性服务

查看访问统计

相关权益政策

暂无数据

收藏/分享

除非特别说明，本系统中所有内容都受版权保护，并保留所有权利。