AKR1C3, as a novel target of DAB2IP signaling, maintains the protein stability of androgen receptor splicing variant in prostate cancer | |
Wu, Kaijie; Wang, Bin; Hsieh, Jer-Tsong; He, Dalin | |
刊名 | INTERNATIONAL JOURNAL OF UROLOGY |
2018 | |
卷号 | 25页码:211-211 |
ISSN号 | 0919-8172 |
URL标识 | 查看原文 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/2830102 |
专题 | 西安交通大学 |
推荐引用方式 GB/T 7714 | Wu, Kaijie,Wang, Bin,Hsieh, Jer-Tsong,et al. AKR1C3, as a novel target of DAB2IP signaling, maintains the protein stability of androgen receptor splicing variant in prostate cancer[J]. INTERNATIONAL JOURNAL OF UROLOGY,2018,25:211-211. |
APA | Wu, Kaijie,Wang, Bin,Hsieh, Jer-Tsong,&He, Dalin.(2018).AKR1C3, as a novel target of DAB2IP signaling, maintains the protein stability of androgen receptor splicing variant in prostate cancer.INTERNATIONAL JOURNAL OF UROLOGY,25,211-211. |
MLA | Wu, Kaijie,et al."AKR1C3, as a novel target of DAB2IP signaling, maintains the protein stability of androgen receptor splicing variant in prostate cancer".INTERNATIONAL JOURNAL OF UROLOGY 25(2018):211-211. |
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