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Naringin inhibits high glucose-induced cardiomyocyte apoptosis by attenuating mitochondrial dysfunction and modulating the activation of the p38 signaling pathway
Huang, Haili[1]; Wu, Keng[2]; You, Qiong[2]; Huang, Ruina[2]; Li, Shanghai[2]; Wu, Keng[2]
刊名INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE
2013
卷号32页码:396-402
关键词naringin cardiomyocyte apoptosis mitochondrial dysfunction p38
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/2222322
专题华南理工大学
作者单位1.[1]Guangdong Med Coll, Affiliated Hosp, Clin Res Ctr, Zhanjiang 524001, Guangdong, Peoples R China
2.[2]Guangdong Med Coll, Affiliated Hosp, Dept Cardiol, Zhanjiang 524001, Guangdong, Peoples R China
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GB/T 7714
Huang, Haili[1],Wu, Keng[2],You, Qiong[2],et al. Naringin inhibits high glucose-induced cardiomyocyte apoptosis by attenuating mitochondrial dysfunction and modulating the activation of the p38 signaling pathway[J]. INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE,2013,32:396-402.
APA Huang, Haili[1],Wu, Keng[2],You, Qiong[2],Huang, Ruina[2],Li, Shanghai[2],&Wu, Keng[2].(2013).Naringin inhibits high glucose-induced cardiomyocyte apoptosis by attenuating mitochondrial dysfunction and modulating the activation of the p38 signaling pathway.INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE,32,396-402.
MLA Huang, Haili[1],et al."Naringin inhibits high glucose-induced cardiomyocyte apoptosis by attenuating mitochondrial dysfunction and modulating the activation of the p38 signaling pathway".INTERNATIONAL JOURNAL OF MOLECULAR MEDICINE 32(2013):396-402.
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