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	Aberrant subcellular localization of SQSTM1/p62 contributes to increased vulnerability to proteotoxic stress recovery in Huntington's disease
	Huang Ningjing[1]; Erie Christine[2]; Lu Michael L[3]; Wei Jianning[4]
刊名	Molecular and cellular neurosciences
	2018
卷号	88页码:43-52
关键词	Huntington's disease Protein aggregates Proteotoxicity SQSTM1/p62 Ubiquitin
ISSN号	1095-9327
URL标识	查看原文
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/2181452
专题	上海大学
作者单位	1.[1]Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton 33431, FL, USA 2.Department of Neurology, Shanghai Municipal Hospital of Traditional Chinese Medicine, Shanghai University of Traditional Chinese Medicine, Shanghai 200071, China. 3.[2]Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton 33431, FL, USA. 4.[3]Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton 33431, FL, USA. 5.[4]Department of Biomedical Science, Charles E. Schmidt College of Medicine, Florida Atlantic University, Boca Raton 33431, FL, USA. Electronic address: jwei@health.fau.edu.
推荐引用方式 GB/T 7714	Huang Ningjing[1],Erie Christine[2],Lu Michael L[3],et al. Aberrant subcellular localization of SQSTM1/p62 contributes to increased vulnerability to proteotoxic stress recovery in Huntington's disease[J]. Molecular and cellular neurosciences,2018,88:43-52.
APA	Huang Ningjing[1],Erie Christine[2],Lu Michael L[3],&Wei Jianning[4].(2018).Aberrant subcellular localization of SQSTM1/p62 contributes to increased vulnerability to proteotoxic stress recovery in Huntington's disease.Molecular and cellular neurosciences,88,43-52.
MLA	Huang Ningjing[1],et al."Aberrant subcellular localization of SQSTM1/p62 contributes to increased vulnerability to proteotoxic stress recovery in Huntington's disease".Molecular and cellular neurosciences 88(2018):43-52.

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