Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis

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	Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis
	Lin, CJ; Ho, HY; Cheng, ML; Cheng, TH; Yu, JS; Chiu, DTY
刊名	FREE RADICAL BIOLOGY AND MEDICINE
	2010-08-01
卷号	49 期号:3 页码:361-373
关键词	G6PD Oxidative stress Dephosphorylation MKP-1 Apoptosis
ISSN号	0891-5849
通讯作者	Yu, JS (reprint author), Chang Gung Univ, Grad Inst Med Biotechnol, Tao Yuan 333, Taiwan.
学科主题	Biochemistry & Molecular Biology; Endocrinology & Metabolism
语种	英语
WOS记录号	WOS:000279645700007
内容类型	期刊论文
源URL	[http://ir.lzu.edu.cn/handle/262010/114884]
专题	生命科学学院_期刊论文
推荐引用方式 GB/T 7714	Lin, CJ,Ho, HY,Cheng, ML,et al. Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis[J]. FREE RADICAL BIOLOGY AND MEDICINE,2010,49(3):361-373.
APA	Lin, CJ,Ho, HY,Cheng, ML,Cheng, TH,Yu, JS,&Chiu, DTY.(2010).Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis.FREE RADICAL BIOLOGY AND MEDICINE,49(3),361-373.
MLA	Lin, CJ,et al."Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis".FREE RADICAL BIOLOGY AND MEDICINE 49.3(2010):361-373.