Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis | |
Lin, CJ; Ho, HY; Cheng, ML; Cheng, TH; Yu, JS; Chiu, DTY | |
刊名 | FREE RADICAL BIOLOGY AND MEDICINE
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2010-08-01 | |
卷号 | 49期号:3页码:361-373 |
关键词 | G6PD Oxidative stress Dephosphorylation MKP-1 Apoptosis |
ISSN号 | 0891-5849 |
通讯作者 | Yu, JS (reprint author), Chang Gung Univ, Grad Inst Med Biotechnol, Tao Yuan 333, Taiwan. |
学科主题 | Biochemistry & Molecular Biology; Endocrinology & Metabolism |
语种 | 英语 |
WOS记录号 | WOS:000279645700007 |
内容类型 | 期刊论文 |
源URL | [http://ir.lzu.edu.cn/handle/262010/114884] ![]() |
专题 | 生命科学学院_期刊论文 |
推荐引用方式 GB/T 7714 | Lin, CJ,Ho, HY,Cheng, ML,et al. Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis[J]. FREE RADICAL BIOLOGY AND MEDICINE,2010,49(3):361-373. |
APA | Lin, CJ,Ho, HY,Cheng, ML,Cheng, TH,Yu, JS,&Chiu, DTY.(2010).Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis.FREE RADICAL BIOLOGY AND MEDICINE,49(3),361-373. |
MLA | Lin, CJ,et al."Impaired dephosphorylation renders G6PD-knockdown HepG2 cells more susceptible to H2O2-induced apoptosis".FREE RADICAL BIOLOGY AND MEDICINE 49.3(2010):361-373. |
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